编号	英文名称	中文名称
JL-FA-01	Amebiasis (AME)	阿米巴病
JL-FA-02	Allergens, Rast scores	过敏原，放射性过敏原吸收实验。指对特定的人群引起免疫反应或者过敏反应的食品中的蛋白质
JL-FA-03	Allergens, Rast scores negative	过敏原，放射性过敏原吸收实验阴性
JL-FA-04	Anti-cyclic citrullinated peptide Antibody (CCP) Arthritis	抗环瓜氨酸肽抗体
JL-FA-05	ASCA Saccharomyces Cerevi	人抗酿酒酵母抗体（ASCA）
JL-FA-06	Aspergillis	麴菌病
JL-FA-07	Beta 2 Glycoprotein	β2糖蛋白
JL-FA-08	Beta 2 Glycoprotein IgM	β2糖蛋白 IGM
JL-FA-09	Bordela Pertussis	百日咳杆菌
JL-FA-10	Bordela Pertussis IgM	百日咳杆菌 IGM
JL-FA-11	C-ANCA	C-抗中性粒细胞胞浆抗体（ANCA)
JL-FA-12	Cardiolipin	心肌磷脂
JL-FA-13	Cardiolipin IgA	心肌磷脂 IGA
JL-FA-14	Cardiolipin IgG	心肌磷脂 IGG
JL-FA-15	Cardiolipin IgM	心肌磷脂 IGM
JL-FA-16	Cerebral Spinal Fluid	脑脊髓液
JL-FA-17	Chagas	恰加斯病/南美锥虫
JL-FA-18	Chlamydia	衣原体
JL-FA-19	Chlamydia IgA	衣原体IGA
JL-FA-20	Chlamydia IgG	衣原体IGG
JL-FA-21	Chlamydia IgM	衣原体IGM
JL-FA-22	Chlamydia Neg	衣原体阴性
JL-FA-23	Clotting Factor C3	凝固因子C3
JL-FA-24	Clotting Factor C4	凝固因子C4
JL-FA-25	Coccidiodes	球孢菌
JL-FA-26	Cytomegalovirus (CMV) Neg	巨细胞病毒抗体阴性
JL-FA-27	CMV IgG	巨细胞病毒 IGG阳性
JL-FA-28	CMV IgM VCA	巨细胞病毒 IGM 阳性
JL-FA-29	C-Reactive Protein (CRP)	C-反应蛋白质
JL-FA-30	Dengue Fever	登革热
JL-FA-31	Dengue Fever IgM	登革热 IGM
JL-FA-32	DS (Double Stranded) DNA	双链脱氧核糖核酸
JL-FA-33	EBNA (Epstein-Barr nuclear antigen) IgG	EB病毒核抗原 IGG
JL-FA-34	EBNA (Epstein-Barr nuclear antigen) IgM	EB病毒核抗原 IGM
JL-FA-35	Epstein Barr Virus (EBV) Negative Plasma	EB病毒阴性血浆
JL-FA-36	Epstein Barr Virus (EBV) EA IgM	EB病毒早期抗原 IGM
JL-FA-37	Epstein Barr Virus (EBV) VCA IgM	EB病毒壳蛋白 IGM
JL-FA-38	Epstein Barr Virus (EBV) EA IgG	EB病毒早期抗原 IGG
JL-FA-39	EMA (Endomysial Antibodies)	肌内膜
JL-FA-40	Gliadin	麸蛋白,麦醇溶蛋白,麦胶蛋白
JL-FA-41	Gliadin IgG	麦醇溶蛋白 IGG
JL-FA-42	Gliadin IgA	麦醇溶蛋白 IGA
JL-FA-43	Glomerular Basement Membrane (GBMA)	肾小球基底膜病
JL-FA-44	Helicobacter pylori IgA	幽门螺旋杆菌IGA
JL-FA-45	Helicobacter pylori IgG	幽门螺旋杆菌IGG
JL-FA-46	Helicobacter pylori IgM	幽门螺旋杆菌IGM
JL-FA-47	Helicobacter pylori Negative	幽门螺旋杆菌阴性
JL-FA-48	Helicobacter pylori Positive Plasma	幽门螺旋杆菌阴性血浆
JL-FA-49	Hepatitis A Virus (HAV) Pos. Plasma	甲型肝炎病毒阳性血浆
JL-FA-50	Hepatitis A Virus (HAV) IgM	甲型肝炎病毒IGM
JL-FA-51	Hepatitis B Core (HBc) IgG	乙型肝炎病毒核心 IGG
JL-FA-52	Hepatitis B Core (HBc) IgM	乙型肝炎病毒核心 IGM
JL-FA-53	Anti Hbe (Antibody to HBV antigen)	乙肝抗体
JL-FA-54	Hepatitis Delta Virus	丁型肝炎病毒
JL-FA-55	HBeAg (HBV e antigen)	乙肝 E抗原
JL-FA-56	anti-HBs (HBV surface antibody)	乙肝表面抗体
JL-FA-57	Hepatitis B (HBsAg) "Chronic"	乙型肝炎（乙肝表面抗原）“慢性病
JL-FA-58	HBsAg (HBV surface antigen) Serum	乙肝表面抗原血清
JL-FA-59	HBsAg (AD)	乙肝表面抗原（AD）
JL-FA-60	HBsAg (AY)	乙肝表面抗原（AY）
JL-FA-61	HBV Positive Plasma	乙肝阳性血浆
JL-FA-62	HBV DNA Plasma	乙肝DNA血浆
JL-FA-63	HBV DNA Serum	乙肝DNA血清
JL-FA-64	HBV DNA type A	A型乙肝DNA
JL-FA-65	HBV DNA type B	B型乙肝DNA
JL-FA-66	HBV DNA type C	C型乙肝DNA
JL-FA-67	HBV DNA type D	D型乙肝DNA
JL-FA-68	HBV DNA type E	E型乙肝DNA
JL-FA-69	HBV DNA type F	F型乙肝DNA
JL-FA-70	HBV Antibody HCV Antibody Plasma CO-INFECTED	乙肝和丙肝联合感染血浆
JL-FA-71	HCV (Hepatitis C Virus) Antibody	丙型肝炎抗体
JL-FA-72	HCV Core Antigen Positive	丙肝核心抗原阳性
JL-FA-73	HCV RNA PLASMA Genotype 1	基因1型丙肝RNA 血浆
JL-FA-74	HCV RNA PLASMA Genotype 2	基因2型丙肝RNA 血浆
JL-FA-75	HCV RNA PLASMA Genotype 3	基因3型丙肝RNA 血浆
JL-FA-76	HCV RNA PLASMA Genotype 4	基因4型丙肝RNA 血浆
JL-FA-77	HCV RNA PLASMA Genotype 5	基因5型丙肝RNA 血浆
JL-FA-78	HCV RNA PLASMA Genotype 6	基因6型丙肝RNA 血浆
JL-FA-79	HCV Riba single band	丙肝免疫印迹单波段
JL-FA-80	HCV RIBA Pos. (multiple bands)	丙肝免疫印迹阳性多波段
JL-FA-81	HCV Negative	丙肝阴性
JL-FA-82	HCV RNA Pos (quantitative)	丙肝RNA阳性（定量）
JL-FA-83	Hepatitis E	戊型肝炎
JL-FA-84	Herpes Simplex Virus (HSV)1/2 Positive Plasma	单纯性疱疹病毒1/2阳性血浆
JL-FA-85	Herpes Simplex Virus (HSV) 1 Negative Plasma	单纯性疱疹病毒1 阴性血浆
JL-FA-86	Herpes Simplex Virus (HSV) 1 IgG	单纯性疱疹病毒1 IGG
JL-FA-87	Herpes Simplex Virus (HSV 1) IgM	单纯性疱疹病毒1 IGM
JL-FA-88	Herpes Simplex Virus (HSV) 2 IgG	单纯性疱疹病毒2 IGG
JL-FA-89	Herpes Simplex Virus (HSV) 2 IgM	单纯性疱疹病毒2 IGG
JL-FA-90	Histone	组蛋白
JL-FA-91	Human Anti Mouse Ab (HAMA)	人抗鼠抗体
JL-FA-92	Human immunodeficiency virus (HIV) 1 Neg	HIV I 阴性
JL-FA-93	anti Human immunodeficiency virus (HIV) 1 Plasma	抗HIV I 血浆
JL-FA-94	anti Human immunodeficiency virus (HIV) 1 Serum	抗HIV I 血清
JL-FA-95	anti Human immunodeficiency virus (HIV) 2 Western Blot Tested	抗HIV 2 免疫印迹
JL-FA-96	anti Human immunodeficiency virus (HIV) 1/2 2 HIV (+)	抗HIV 1/2 2 HIV阳性
JL-FA-97	Human immunodeficiency virus (HIV) Ag	HIV抗原
JL-FA-98	HIV RNA (quantitative) Plasma	HIV RNA 定量血浆
JL-FA-99	HIV RNA (quantitative) Serum	HIV RNA 定量血清
JL-FA-100	HIV1 Subtype A	HIV1 亚型A
JL-FA-101	HIV1 Subtype B	HIV1 亚型B
JL-FA-102	HIV1 Subtype C	HIV1 亚型C
JL-FA-103	HIV1 Subtype D	HIV1 亚型D
JL-FA-104	HIV1 Subtype E	HIV1 亚型E
JL-FA-105	HIV1 Subtype F	HIV1 亚型F
JL-FA-106	HIV1 Subtype G	HIV1 亚型G
JL-FA-107	HIV1 Subtype H	HIV1 亚型H
JL-FA-108	HIV1 Subtype J	HIV1 亚型J
JL-FA-109	HIV1 Subtype K	HIV1 亚型K
JL-FA-110	HIV1 Group O	HIV1 亚型O
JL-FA-111	Human immunodeficiency virus (HIV) 2 Antibody Plasma	HIV 2 抗体血浆
JL-FA-112	Human immunodeficiency virus (HIV) 2 Antibody Serum	HIV 2 抗体血清
JL-FA-113	HPV (Human Papiloma Virus) Negative	人乳状瘤病毒HPV阴性
JL-FA-114	HPV (Human Papiloma Virus) Positive	人乳状瘤病毒HPV阳性
JL-FA-115	Human immunodeficiency virus (HIV) Antibody HCV Antibody Plasma COINFECTED	HIV 抗体 HCV
JL-FA-116	Human T-cell Lymphotropic Virus (HTLV) I/II	人嗜T淋巴细胞病毒(HTLV) I/II
JL-FA-117	Human T-cell Lymphotropic Virus (HTLV) I	人嗜T淋巴细胞病毒(HTLV) I
JL-FA-118	Human T-cell Lymphotropic Virus (HTLV) II	人嗜T淋巴细胞病毒(HTLV) II
JL-FA-119	Jo-1	多发性肌炎抗原JO-1
JL-FA-120	IgE < 5,000 Ku/L	IgE < 5,000 Ku/L
JL-FA-121	Legionella	军团杆菌属
JL-FA-122	Leptospira	军团杆菌属
JL-FA-123	Lyme Disease	莱姆(氏)病:蜱传播的全身性疾病,常在夏季发生
JL-FA-124	Lyme IgG	莱姆(氏)病 IGG
JL-FA-125	Lyme IgM	莱姆(氏)病 IGM
JL-FA-126	Lyme Disease Neg	莱姆(氏)病阴性
JL-FA-127	Malaria	疟疾
JL-FA-128	Mononucleosis (infectious)	单核细胞增多症（有传染性的）
JL-FA-129	Mononucleosis Negative	单核细胞增多症阴性
JL-FA-130	Measles Negative	麻疹阴性
JL-FA-131	Measles IgG	麻疹 IGG
JL-FA-132	Measles IgM	麻疹 IGM
JL-FA-133	Microsomal Anti-thyroid peroxidase antibody (TPO) Positive Plasma Standard Titer (typically 1,000-3,000 IU/mL)	微粒体抗甲状腺过氧化物酶抗体
JL-FA-134	Microsomal Anti-thyroid peroxidase antibody (TPO) Negative Plasma	微粒体抗甲状腺过氧化物酶抗体
JL-FA-135	Anti-mitochondrial antibody (AMA)	抗线粒体抗体
JL-FA-136	Multiple Sclerosis	多发性硬化症
JL-FA-137	Mumps IgG	流行性腮腺炎 IGG
JL-FA-138	Mumps Ab IgM	流行性腮腺炎抗体 IGM
JL-FA-139	Mumps Antibody Negative Plasma	流行性腮腺炎抗体阴性血浆
JL-FA-140	Mumps Antibody Negative Serum	流行性腮腺炎抗体阴性血清
JL-FA-141	Myeloma Plasma	骨髓瘤血浆
JL-FA-142	Myeloma IgA	骨髓瘤IGA
JL-FA-143	Myeloma IgE	骨髓瘤IGE
JL-FA-144	Myeloma IgG	骨髓瘤IGG
JL-FA-145	Myeloma IgM	骨髓瘤IGM
JL-FA-146	Mycoplasma	支原体
JL-FA-147	Mycoplasma Negative	支原体阴性
JL-FA-148	Mycoplasma IgG	支原体IGG
JL-FA-149	Mycoplasma IgM	支原体IGM
JL-FA-150	Mycoplasma PCR	支原体PCR
JL-FA-151	Normal Human Plasma	正常人血浆
JL-FA-152	Normal Human Serum	正常人血清
JL-FA-153	Nuclear Antibody Centromere	核抗体着丝粒
JL-FA-154	Nuclear Antibody, Speckled ANA	核抗体，斑点抗核抗体
JL-FA-155	Nuclear Antibody, Nucleolar ANA	核抗体,核仁抗核抗体
JL-FA-156	Nuclear Antibody, Homogeneous ANA	核抗体，同质抗核抗体
JL-FA-157	Nuclear Antiobody, Speckled. (ANA) Negative	核抗体，斑点。抗核抗体阴性
JL-FA-158	P-ANCA (associated neutrophil cytoplasmic antibodies)	相关的嗜中性粒细胞胞浆抗体
JL-FA-159	Parietal Cell Antibody (PCA)	胃)壁细胞抗体
JL-FA-160	Parvo positive plasma	细小病毒阳性血浆
JL-FA-161	Parvo IgM	细小病毒 IGM
JL-FA-162	Parvo IgG	细小病毒 IGG
JL-FA-163	Parvo Negative Plasma	细小病毒阴性血浆
JL-FA-164	Parvo DNA positive	细小病毒 DNA 阳性
JL-FA-165	Phospholipid Positive Plasma	磷脂阳性血浆
JL-FA-166	Prothrombin	凝血酶原,凝血因子
JL-FA-167	Rheumatoid Factor (RF) <1000 IU/mL	类风湿因子<1000 IU/mL
JL-FA-168	Rheumatoid Factor (RF) 1001-2000 IU/mL	类风湿因子1001-2000 IU/mL
JL-FA-169	Rheumatoid Factor (RF) 2001-4000 IU/mL	类风湿因子 2001-4000 IU/mL
JL-FA-170	Rheumatoid Factor (RF) 4001-5000 IU/mL	类风湿因子 4001-5000 IU/mL
JL-FA-171	Rheumatoid Factor (RF) >5000 IU/mL	类风湿因子>5000 IU/mL
JL-FA-172	Ribonucleoprotein (RNP) Positive	核糖核蛋白阳性
JL-FA-173	Rubella Chimeric	风疹
JL-FA-174	Rubella Negative	风疹阴性
JL-FA-175	Rubella IgG	风疹IGG
JL-FA-176	Rubella IgM	风疹IGM
JL-FA-177	Rubeola Negative Plasma	风疹阴性血浆
JL-FA-178	Rubeola IgG	风疹IGG
JL-FA-179	Scleroderma (Scl-70) Pos	胶原沉着病,硬皮病,硬皮症阳性
JL-FA-180	Scleroderma (Scl-70) Negative	硬皮病阴性
JL-FA-181	Sickle Cell Fresh Whole Blood	镰刀形红细胞新鲜全血
JL-FA-182	Smith (SM)	抗Smith抗体阳性血清（SLE的特征性抗体）
JL-FA-183	SMITH RNP	抗RNP抗体阳性血清（SLE的特征性抗体）
JL-FA-184	Smooth Muscle (ASMA)	抗平滑肌抗体阳性血清
JL-FA-185	Sjogren syndrome antigen A (SSA) Positive	舍格伦综合征或干燥综合征抗原A 阳性
JL-FA-186	Sjogren syndrome antigen B (SSB) Positive	舍格伦综合征抗原B 阳性
JL-FA-187	Sjogren syndrome antigen B (SSB) Negative	舍格伦综合征抗原B阴性
JL-FA-188	Streptolysin O Ab (ASO)	链球菌溶血素O抗体
JL-FA-189	Syphilis (RPR - Rapid Plasma Reagin) Positive Plasma	梅毒（梅毒-快速血浆反应）阳性血浆
JL-FA-190	Syphilis (RPR - Rapid Plasma Reagin) Negative Plasma	梅毒（梅毒-快速血浆反应）阴性血浆
JL-FA-191	Syphilis/ATA/T. pallidum IgG	梅毒ATA/T，苍白球IGG
JL-FA-192	Syphilis/ATA/T. pallidum IgM	梅毒ATA/T，苍白球IGM
JL-FA-193	Systemic Lupus Erythematosus (SLE) Positive	全身性红斑狼疮阳性
JL-FA-194	Systemic Lupus Erythematosus (SLE) Negative	全身性红斑狼疮阴性
JL-FA-195	TG/TPO Positive (Standard Titer 1,000 - 3000 IU/mL)	甲状腺球蛋白/甲状腺过氧化物酶阳性
JL-FA-196	TG/TPO Negative	甲状腺球蛋白/甲状腺过氧化物酶阴性
JL-FA-197	TTG (Tissue Transglutaminase)	组织转谷氨酰胺酶
JL-FA-198	TTG (Tissue Transglutaminase) IgA	组织转谷氨酰胺酶 IGA
JL-FA-199	ToRCH (Toxo, Rubella, CMV, HSV) Positive	优生优育（弓形虫，风疹，巨细胞，单胞）阳性
JL-FA-200	ToRCH (Toxo, Rubella, CMV, HSV) Negative	优生优育（弓形虫，风疹，巨细胞，单胞）阴性
JL-FA-201	Toxoplasmosis (Toxo)	弓形虫病
JL-FA-202	Toxoplasmosis (Toxo) IgG	弓形虫病IGG
JL-FA-203	Toxoplasmosis (Toxo) IgM	弓形虫病IGM
JL-FA-204	Thyroglobulin (TG) Positive Plasma	甲状腺球蛋白阳性血浆
JL-FA-205	Thyroglobulin (TG) Negative	甲状腺球蛋白阴性
JL-FA-206	Varicella-Zoster Virus (VZV) Negative	水痘-带状疱疹病毒阴性
JL-FA-207	Varicella-Zoster Virus (VZV) IgG	水痘-带状疱疹病毒IGG
JL-FA-208	Varicella-Zoster Virus (VZV) IgM	水痘-带状疱疹病毒IGM
JL-FA-209	West Nile Virus (WNV)	西尼罗河脑炎病毒
JL-FA-210	West Nile Virus (WNV) IgM	西尼罗河脑炎病毒IGM

美国Seracare

此次研究中，研究人员通过导入这个基因的拷贝，实现了该受体的过量表达。而实际生活中，并不能将这一做法应用到人身上，因而，研究人员将进一步开发靶向这一基因的药物。
一项来自美国俄亥俄州立大学综合癌症中心的证据显示，经基因修饰的免疫细胞或对多发性骨髓瘤的治疗有效果，且这些细胞可在实验室中培养增殖。该研究成果5月28日刊载在Clinical Cancer Research杂志。
研究人员对人类免疫细胞(来自患者的细胞系和骨髓瘤细胞)——T淋巴细胞进行修饰，靶向存在于大多数(占比95%以上)的骨髓瘤细胞中的CS1分子，以期杀死细胞。研究人员在实验室中培养增殖这些修饰细胞，然后将其注入动物模型中，杀死人骨髓瘤细胞。
与对照组t细胞相比，修饰T细胞可以更好地识别过表达CS1的多发性骨髓瘤细胞，并激活。注射修饰T细胞的所有小鼠在接受治疗后存活了44天，而在另外两个对照组中，这一数据只有29%和17%。
这种疗法的一个重要的优势是，这些治疗T细胞在体内有复制潜能，所以或许可以抑制肿瘤生长，长时间内避免复发。
墨尔本的研究人员证明，A型白血病能成功地‘逆转’通过诱骗癌症细胞回到正常发育状态。这一发现使用B-前体急性淋巴细胞白血病(B-ALL)模型，是一种zui常见的影响儿童的癌症。研究人员揭示关闭一个称为Pax5的基因在B-ALL模型中能够诱发癌症，而恢复它的功能能够‘治愈’这种疾病。研究发表在《Genes & Development》期刊上。
研究者之一的刘女士说团队利用一种发展的‘基因开关’技术去抑制然后再活化Pax5在白血病模型中。
“随着其它基因变化一起，失活的Pax5驱动正常血细胞变成白血病细胞，之前已经报道过，”刘女士说。“然而我们*揭示重新复活Pax5能够使细胞重新开始其正常发育并丢失那些类似癌症的特性，有效地治疗白血病。吸引我们的是只是回复Pax5就足够使这些癌细胞恢复常态化，尽管其它基因是改变的。”
白血病中，未成熟白血细胞复制异常并在骨髓中累积，干扰正常血细胞的产生。
刘女士说在儿童B-ALL中Pax5常常是一个‘消失’的基因。“Pax5对于一种白血细胞——b细胞正常发育*的，”她说。“当Pax5功能缺乏抵抗力时，发育中的B细胞陷入一种不成熟状态并且变成癌性的。我们证明恢复Pax5功能，即使是在已经发生癌病的细胞中，移除这个‘阻塞’，使得细胞发育成正常白血细胞。”
Dickins博士说这项研究揭示了Pax5的功能，是约100种已知‘抑制’人类肿瘤的基因之一。“当这些肿瘤抑制基因通过改变DNA而被失活时，肿瘤开始发育，”Dickins博士说。

美国Seracare

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【公司名称】 广州健仑生物科技有限公司
【】杨永汉
【】
【腾讯】 2042552662
【公司地址】 广州清华科技园创新基地番禺石楼镇创启路63号二期2幢101-3室
【企业文化】

In this study, researchers introduced an overexpression of this gene by introducing a copy of this gene. And in real life, this practice can not be applied to people, so the researchers will further develop drugs that target this gene.
Evidence from the Ohio State University Comprehensive Cancer Center shows that genetically modified immune cells or treatments for multiple myeloma can be effective and that these cells can be cultured and proliferated in the laboratory. The research was published May 28 in Clinical Cancer Research.
Researchers have modified human immune cells (cell lines from patients and myeloma cells), T-lymphocytes, to target CS1 molecules present in the majority (over 95%) of myeloma cells in order to kill cell. The researchers cultured and cultured these modified cells in a laboratory and injected them into animal models to kill human myeloma cells.
Modification of T cells allows better identification and activation of CS1-overexpressing multiple myeloma cells as compared to control t-cells. All mice injected with modified T cells survived for 44 days after treatment, compared with only 29% and 17% of the other two controls.
An important advantage of this therapies is that these therapeutic T cells have the potential to replicate in the body and so may be able to inhibit tumor growth and avoid relapse for long periods of time.
Researchers in Melbourne have demonstrated that type A leukemia can successfully 'reverse' by inducing cancer cells to return to their normal developmental state. This finding, using the B-ALL acute lymphoblastic leukemia (B-ALL) model, is one of the most common cancers in children. Researchers have revealed that the closure of a gene called Pax5 is capable of inducing cancer in the B-ALL model, and restoring its function to 'cure' the disease. Research published in "Genes & Development" journal.
Ms. Liu, one of the researchers, said the team took advantage of a newly developed 'gene switch' technique to repress and then reactivate Pax5 in leukemia models.
"As other genetic changes, inactivated Pax5 drives normal blood cells to become leukemic cells, as previously reported," Ms. Liu said. "However, for the first time, we have shown that resuscitation of Pax5 allows cells to restart their normal development and lose those cancers-like properties that effectively treat leukemias, and it is enough to appeal to Pax5 that these cells are restored to normalization, although other genes are changed."
In leukemia, immature white blood cells replicate abnormally and accumulate in the bone marrow, interfering with the production of normal blood cells.
Ms. Liu said Pax5 is often a 'vanishing' gene in children with B-ALL. "Pax5 is essential for the normal development of a white blood cell-b cell," she said. "When Pax5 function lacks resistance, developing B cells plunge into an immature state and become cancerous.We show that restoring Pax5 function, even in cells where cancer has already occurred, removes this 'obstruction' , Making the cells develop into normal white blood cells. "
Dr. Dickins said the study revealed the function of Pax5 and is one of about 100 genes known to 'suppress' human tumors. "Tumors begin to develop when these tumor suppressor genes are inactivated by changing DNA," says Dr. Dickins.

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